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POTENTIAL MECHANISMS UNDERLYING INFECTION-ASSOCIATED DYSAUTONOMIA & POTS The autonomic nerves or autonomic ganglia may be affected by: Chronic infection Autoimmune response triggered by an infection Damage to autonomic nerves by an infection Neuro-inflammation triggered by debris left-over after an acute infection is treated CHRONIC ILLNESSES ASSOCIATED WITH POTS Post Treatment Lyme Disease Long COVID Myalgic Encephalomyelitis/Chronic Fatigue Syndrome or ME/CFS Sjogrens Syndrome Systemic Lupus Erythematosus Hashimotos Thyroiditis Celiac Disease Diabetes MS Other Infection Associated Illnesses RESEARCH NEWS Review Article by Brit Adler, MD, in Frontiers in Neurology: Dysautonomia following Lyme disease: a key component of post-treatment Lyme disease syndrome

632 Di-Huang-Yin-Zi effectively decreases ROS and MDA levels in prefrontal cortex, increases the expression of ferroptosis-related markers GPX4 and SLC7A11 and, and decreases iron deposition, thereby improving depressive symptoms and synaptic ultrastructure of cortical regions in post-stroke depressed (PSD) rats, suggesting that Di-Huang-Yin-Zi can reduce the symptoms and enhance function in PSD rats by inhibiting ferroptosis through SLC7A11/GPX4 pathway

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